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While most of the somatically synthesized GluA1 follows the canonical Golgi-dependent secretory pathway 17 , in dendrites, locally synthesized GluA1 traffics through the ER-Golgi intermediary compartment (ERGIC) into a Golgi-independent pathway, accumulating in Rab11-positive recycling endosomes prior to PM insertion 18

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Similar to Fe 3+ , Cu 2+ binds to A β and elicits similar response in terms of activation of A β aggregation, H 2 O 2 generation and other oxidative stress-mediated neuronal toxicity [215, 218, 219]