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The pro-apoptotic action involves mitochondrial dysfunction and activation of caspases, while regulating many pathways such as epithelial-mesenchymal transition (EMT) and oxidative stress, which may also play roles in neuronal protection and survival [49]

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They have neuroprotective effects in humans, reducing disease progression by protecting neurons from oxidative damage, Aβ-induced neuronal injury and neurotoxicity, and nitric oxide-induced toxicity while increasing neuronal function and regeneration and modulating neuronal signaling pathways [20,28]