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The goal here was to use combinatorial chemistry to create single molecules with multi-receptor potency comparable to the native incretins that might produce large effect sizes in preclinical studies such that effects were likely to be translatable to humans

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Both strains have: rfa mutations, a defective lipopolysaccharide layer that makes bacteria more permeable to larger molecules uvrB mutations, which eliminate excision repair of DNA damage the pKM101 plasmid, which increases error-prone repair of DNA damage Why is it important to investigate mutagenicity of compounds